Modulation of the complement system in monocytes contributes to tuberculosis-associated immune reconstitution inflammatory syndrome

H.T.T. Tran; R. Van den Bergh; M. Massinga Loembe; W. Worodria; H. Mayanja-Kizza; R. Colebunders; F. Mascart; P. Stordeur; L. Kestens; P. De Baetselier; G. Raes; TB-IRIS Study Group

doi:10.1097/QAD.0b013e328361648b

Modulation of the complement system in monocytes contributes to tuberculosis-associated immune reconstitution inflammatory syndrome

H.T.T. Tran, R. Van den Bergh, M. Massinga Loembe, W. Worodria, H. Mayanja-Kizza, R. Colebunders, F. Mascart, P. Stordeur, L. Kestens, P. De Baetselier, G. Raes, TB-IRIS Study Group

Research output: Contribution to journal › A1: Web of Science-article › peer-review

Original language	English
Journal	AIDS
Volume	27
Issue number	11
Pages (from-to)	1725-1734
Number of pages	10
ISSN	0269-9370
DOIs	https://doi.org/10.1097/QAD.0b013e328361648b
Publication status	Published - 2013

Keywords

Viral diseases
HIV
AIDS
Associations
Tuberculosis
Mycobacterium tuberculosis
Immune reconstitution inflammatory syndrome (IRIS)
IRIS
Disease progression
Monocytes
HAART
Antiretrovirals
Complement
Inflammation

Access to Document

10.1097/QAD.0b013e328361648b

http://lib.itg.be/pdf/itg/2013/2013aids1725.pdf

Cite this

Tran, H. T. T., Van den Bergh, R., Massinga Loembe, M., Worodria, W., Mayanja-Kizza, H., Colebunders, R., Mascart, F., Stordeur, P., Kestens, L., De Baetselier, P., Raes, G., & Group, TB.-IRIS. S. (2013). Modulation of the complement system in monocytes contributes to tuberculosis-associated immune reconstitution inflammatory syndrome. AIDS, 27(11), 1725-1734. https://doi.org/10.1097/QAD.0b013e328361648b

@article{a3009a8f58814f1c984b0a04f923239f,

title = "Modulation of the complement system in monocytes contributes to tuberculosis-associated immune reconstitution inflammatory syndrome",

keywords = "Viral diseases, HIV, AIDS, Associations, Tuberculosis, Mycobacterium tuberculosis, Immune reconstitution inflammatory syndrome (IRIS), IRIS, Disease progression, Monocytes, HAART, Antiretrovirals, Complement, Inflammation",

author = "H.T.T. Tran and {Van den Bergh}, R. and {Massinga Loembe}, M. and W. Worodria and H. Mayanja-Kizza and R. Colebunders and F. Mascart and P. Stordeur and L. Kestens and {De Baetselier}, P. and G. Raes and Group, {TB-IRIS Study}",

note = "NPP; ITG-B3B; ITG-C6A; ITG-B9A; MULTI; DBM; U-IMMUN; DCS; U-HIVCLI; JIF; DOI; PDF; DSPACE56",

year = "2013",

doi = "10.1097/QAD.0b013e328361648b",

language = "English",

volume = "27",

pages = "1725--1734",

journal = "AIDS",

issn = "0269-9370",

publisher = "Lippincott Williams & Wilkins",

number = "11",

}

Tran, HTT, Van den Bergh, R, Massinga Loembe, M, Worodria, W, Mayanja-Kizza, H, Colebunders, R, Mascart, F, Stordeur, P, Kestens, L, De Baetselier, P, Raes, G & Group, TB-IRISS 2013, 'Modulation of the complement system in monocytes contributes to tuberculosis-associated immune reconstitution inflammatory syndrome', AIDS, vol. 27, no. 11, pp. 1725-1734. https://doi.org/10.1097/QAD.0b013e328361648b

TY - JOUR

T1 - Modulation of the complement system in monocytes contributes to tuberculosis-associated immune reconstitution inflammatory syndrome

AU - Tran, H.T.T.

AU - Van den Bergh, R.

AU - Massinga Loembe, M.

AU - Worodria, W.

AU - Mayanja-Kizza, H.

AU - Colebunders, R.

AU - Mascart, F.

AU - Stordeur, P.

AU - Kestens, L.

AU - De Baetselier, P.

AU - Raes, G.

AU - Group, TB-IRIS Study

N1 - NPP; ITG-B3B; ITG-C6A; ITG-B9A; MULTI; DBM; U-IMMUN; DCS; U-HIVCLI; JIF; DOI; PDF; DSPACE56

PY - 2013

Y1 - 2013

KW - Viral diseases

KW - HIV

KW - AIDS

KW - Associations

KW - Tuberculosis

KW - Mycobacterium tuberculosis

KW - Immune reconstitution inflammatory syndrome (IRIS)

KW - IRIS

KW - Disease progression

KW - Monocytes

KW - HAART

KW - Antiretrovirals

KW - Complement

KW - Inflammation

U2 - 10.1097/QAD.0b013e328361648b

DO - 10.1097/QAD.0b013e328361648b

M3 - A1: Web of Science-article

C2 - 23807270

SN - 0269-9370

VL - 27

SP - 1725

EP - 1734

JO - AIDS

JF - AIDS

IS - 11

ER -